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Examining how GHS-R1a-driven calcium mobilisation potentiates GHRH-induced cAMP-PKA signalling exclusively in somatotrophs without off-target lactotroph and corticotroph co-stimulation, enabling attribution of all observed downstream biological effects to the GH secretory response rather than to co-secreted prolactin or cortisol

(3) promotion of multicenter clinical trials to build an evidence base through evidence-based medicine

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The 200-fold-greater potency of LR3 versus insulin in cell-culture systems coupled with the molecules resistance to enzymatic degradation in serum-free media underlies its adoption as a substitute for native IGF-1 in animal-component-free cell-culture protocols

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