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Additional information Handling Editor: Zhenggao Xiao
10.1111/j.1463-1326.2010.01350.x 85
Disclosure Professor Xiao-Yu Tian reports grants from Clinical Medical Excellence Training Program funded by government (ZF2023112), grants from the 14th Five-Year Plan Clinical Medicine Innovation Research Team of Hebei Medical University, Class B Team Project (2022LCTD-B15), grants from Hebei Provincial Health Commission Project (20230594), grants from Hebei Province Third Batch of Young Top Talents Program, outside the submitted work
Thus, as far as mitochondrial oxidative stress homeostasis is concerned, mGSH is a vital factor maintaining mitochondrial function, controlling cell survival/death, and its depletion causes cell damage and facilitates cell death [57, 59]
inflammation of the pancreas (pancreatitis)
