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When tissue folate and cobalamin stores are intact, an abnormal increment of postmethionine-load homocysteinemia may also reveal a saturation of the transsulfuration pathway caused by dietary methionine excess that might be combined with transsulfuration defects (in cases with basal hyperhomocysteinemia) or with the homozygous thermolabile MTHFR defect (in cases with normal fasting homocysteine) (Cattaneo, 1999)

Enhance Performance and Decrease Recovery Time

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In: Pandey A, Venkata Mohan S, Chang J-S, Hallenbeck PC, Larroche C (Eds) Biomass, Biofules, Biochemicals

Impact of l-carnitine supplementation on metabolic profiles in premature infants