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In these atypical hyperphenylalaninemias, that result from QDPR mutations, restriction of phenylalanine from the diet has no therapeutic benefits

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HUBERMAN: And this is actually going to be a pretty quick discussion because really the main one in this category, besides thymus and beta-4, remember thymus and beta-4 we talked about earlier, this is a peptide that's naturally released from the thymus and the thymus is a structure that depletes over time as children age

This mechanism raises the critical discontinuation question - does stopping GHK-Cu immediately halt enhanced collagen production returning synthesis rates to pre-treatment baseline, or does the improved dermal environment created during treatment continue supporting elevated collagen production through positive feedback mechanisms where better matrix structure itself promotes ongoing synthesis even without continuous peptide stimulation

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