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These medications are designed to help with weight loss and blood sugar control, not to replace thyroid hormone or treat an underactive thyroid

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In mouse models, dual agonism had superior efficacy for weight loss and glucose control compared to a GLP-1RA alone 9 , with additive effects of the GIPR proposed to act through signaling in beta cells 10 , alpha cells 11 , the CNS (central nervous system) 12,13 and adipocytes 14

Importantly, neuroinflammatory processes are closely intertwined with oxidative stress and mitochondrial dysfunction, as inflammatory mediators increase reactive oxygen species production and compromise mitochondrial bioenergetics, further amplifying neuronal vulnerability (33)