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The ANT crystal structures in both cytoplasmic-open (c-state) and matrix-open states (m-state) locked by two inhibitors bound at the solute binding site, carboxyatractyloside and bongkrekic acid respectively, highlighted a conserved alternating-access transport mechanism 5 enabled by the conformational change upon solute binding at the central cavity 6,7
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specific to drug product concentration and allowable levels of denaturation
During cerebral ischemia, the integrity of the BBB is impaired and free iron from circulation enters the brain
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