Indeed, 4-en-VPA is transformed through -oxidation to reactive intermediates such as 2-propyl-2, 4-pentadienoic acid (2, 4-dien-VPA) that are capable of depleting mitochondrial GSH, as suggested by rat studies [60]
The peptide's stability profile allows for both oral and parenteral administration, with documented biological activity through multiple delivery routes including subcutaneous, intramuscular, intraperitoneal, and oral administration
This further helps in regeneration of damaged nerves in the body
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