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While the long-term safety of ipamorelin has yet to be evaluated, we can surmise that, as a growth hormone (GH) secretagogue, long-term ipamorelin therapy may indirectly produce side effects associated with elevated GH levels
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Mechanistically, SLC25A39 deficiency induced mitochondrial dysfunction, characterized by excessive mitochondrial reactive oxygen species (ROS), reduced membrane potential, diminished adenosine triphosphate (ATP) synthesis, aberrant mitochondrial permeability transition pore (mPTP) opening, and cytochrome c release
