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GSH is connected to the hepatotoxicity of acetaminophen because it is used by the liver to bind a toxic byproduct (NAPQI) of acetaminophen metabolism

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4A, Fig

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EGCG can probably, maintain the expression of ISGs and IFN-1 signaling cascade components

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In a burn rat model, intraperitoneal injection of glycine supplementation alleviated severe burn-induced cardiac injury by improving cardiomyocyte energy metabolism and increasing ATP and GSH contents [108]

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By elucidating these mechanisms, this review seeks to highlight potential targets for antioxidant therapies and lifestyle modifications that could mitigate oxidative stress and improve reproductive outcomes in men with infertility

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