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Conclusions Semaglutide effectively ameliorates cognitive dysfunction in T2DM mice, likely through mechanisms involving the reduction of oxidative stress, inflammation, and neuronal apoptosis
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It was because we knew which children would develop ASD later that we were able to call one group pre-ASD. The other group we knew would not develop ASD because they were neurotypical when they reached three to 10 years of age, said Naviaux
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