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Mitochondria-targeted compounds, such as MitoQ and SkQ derivatives, are being investigated for their ability to localize antioxidant effects precisely where oxidative damage is most profound (Vctor et al., 2009)

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GEP44 activates insulin-independent glucose uptake in muscle tissue via its interactions with Y1-R Despite the positive ISR data and hypothesis that GEP12 would prove superior to GEP44, we noted in in vivo studies (vide infra) that it was considerably inferior to GEP44 in terms of food intake and body weight reduction