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AGT is present in the proximal tubule and concurrent increases in AGT-mRNA/protein in renal tissue homogenates has been explained as indicating that transcriptional activation of the AGT gene would be responsible for renal ANG II increase

Our findings align with previous research where OVA exposure increased the MDA concentration [56]

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J Hum Hypertens

Peptides in general are poor substrates and poor inhibitors of cytochrome P450 enzymes because they are typically degraded by proteases rather than hepatic oxidative metabolism [7]