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c-Met / proliferation concern is real and unresolved: c-Met is the product of the MET proto-oncogene, and aberrant HGF/c-Met signaling drives several cancers
However, because these observations rely solely on immunohistochemistry, they indicate potential involvement rather than providing definitive mechanistic evidence, and molecular validation is required to confirm these pathways
The first site interacts with the glutathione moiety of glutathionylated disulfide substrates
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