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Cellular toxicity of polyglutamine expansion proteins: mechanism of transcription factor deactivation
Nevertheless, since GSH cannot be administered directlybecause of its insufficient absorption, stability and solubilityanalogues have been obtained, through modifications at the -glutamyl, cysteine and glycine moieties, and/or the functionalisation of the central cysteine, that are able to inhibit the activity of GSTs 69,87,105
Reduction of MTT by glutathione S-transferase
doi: 10.18632/aging.101986 293 NakanoKWhitakerJWBoyleDLWangWFiresteinGS
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