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2009 Nov;12(4):189-96
In pressure-overload models, cardiomyocyte acetyl CoA carboxylase 2 deletion preserved fatty acid oxidation and prevented metabolic remodeling, showing that altered substrate handling can modify hypertrophic progression ( Several pathways now connect metabolic flux to canonical hypertrophic signaling
Mechanistically, these agents bind the GLP-1 receptor, enhancing glucose-dependent insulin secretion, slowing gastric emptying, and modulating appetite centers
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Low-impact activities such as swimming, walking, or yoga may be better tolerated than high-impact exercise during the adjustment period
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