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These symptoms progress gradually, but if untreated, could be permanent
This comprehensive laboratory surveillance helps rule out organ toxicity or bone marrow suppression that might occur with extended exposure
Once CXXC5 suppression is fully addressed, additional improvement requires targeting other mechanisms through complementary treatments
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found that these two vectors were highly expressed in myocardial mitochondria and showed that inhibition of DIC and OGC increased mitochondrial ROS and GSH depletion, exacerbating ferroptosis in the myocardium [60]
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