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[1] Other causes include autoimmune conditions, certain infections, and inflammatory states that alter B12 metabolism or binding protein production
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The peptide appears to shift macrophage polarization from pro-inflammatory M1 phenotypes toward anti-inflammatory M2 phenotypes, thereby facilitating tissue repair and regeneration
There is no published human safety or efficacy data, no reproductive- or developmental-toxicology research, no studies in hepatic or renal impairment, and no drug-interaction studies
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