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Clausenamide (Clau) has been shown to protect dopaminergic neurons by directly inhibiting ALOX5 activation and its nuclear translocation, thereby reducing lipid peroxidation and suppressing ferroptosis (253)

These findings informed the creation of a unique formulation first validated through an in-vitro cell culture study, which confirmed its ability to activate GLP-1 pathways, reduce the enzyme responsible for breaking it down, and increase receptor activation

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