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In db/db mice and HG-treated TECs, ectonucleoside triphosphate diphosphohydrolase 5 overexpression inhibited apoptosis to preserve TECs via regulating the protein N-glycosylation rate in ER (83)
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Colocalization analysis is based on five hypotheses: (i) absence of any causal variant influencing either protein expression levels or migraine within the genomic region (H0)
However, researchers now want to know whether these medicines may also help the heart, blood vessels, and inflammation-related risks
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