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[18] Many molecular events discussed alongside AOD9604such as cyclic AMP accumulation, protein kinase A activation, hormone-sensitive lipase phosphorylation, perilipin remodeling, and mitochondrial fatty-acid oxidationbelong to established adipocyte biology
So, rather than being driven by systemic insulin resistance or hyperinsulinemia, phenotype D may result from intrinsic ovarian dysfunction of ovaries or abnormalities in folliculogenesis
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