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An update on oxidative damage to spermatozoa and oocytes

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Recently, we found that semaglutide therapy post-stroke can prevent ischemia-induced neurotoxicity and normalize neurogenesis and proliferation of stem cells in the brain via inhibition of p38 MAPK/MKK/c-Jun activity, regulation of Bcl-2/Bax pathways, and C-raf/ERK/caspase-3, restoration of insulin signaling sensitivity, and normalization of the ERK1 and IRS1 pathways (120)

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Please join us by contacting your elected officials to urge them to halt plans to end coverage of GLP-1 weight-loss medications for TFL beneficiaries