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ROS-mediated oxidative stress promotes mitochondrial dysfunction, fostering a self-reinforcing cycle where increased ROS generation perpetuates lipids, proteins and DNA oxidation, thereby exacerbating cellular injury [231]
Precisely, superoxide anion and hydrogen peroxide have been reported to act as mitogens, thereby promoting the rate of DNA replication and cell proliferation in a variety of cultured cells.9, 10 Nonetheless, in excess amounts, oxygen-derived radicals are very harmful to living organisms
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