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To confirm that the activated receptor remained on the cell surface, we performed a cell surface fluorescence assay, which confirmed the presence of PDGFR on the plasma membrane 60 min post stimulation, when cells were transfected with Nsp3 (EEEV) wt but not when they were treated with other control constructs (Fig
PMID: 82 TehraniFRBehboudi-GandevaniS
The two active forms are produced on separate metabolic pathways and perform completely different functions
And it was found that compound 25 exhibited the strongest inhibition of ferroptosis (EC 50 = 8.6 2.2 nM)
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