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Numerous studies utilizing adenoviral transduction of FAK-related non-kinase (FRNK), an endogenous dominant-negative inhibitor of the FAK FAT domain, showed displacement of FAK from focal adhesions, leading to increased cell rounding, detachment, and apoptosis 18,19
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These peptides may support functions such as immune modulation, muscle growth, fat metabolism, and cellular repair
A key point for interpretation is that cell migration and cell proliferation are related but not identical
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