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[18] LanCL1 has been identified as a binding and functional mediator of LAT8881 activity in neuropathic-pain models, but its role in adipocyte or whole-body metabolic effects remains unresolved

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Another observation from studies on murine models exposed to catabolic agents revealed a reduction in the excretion of N-methylhistidine, a marker of muscle protein degradation, which was notably more pronounced with IGF-1 LR3potentially three times greater than with IGF-I

Early reviews suggest potential benefits and call for more controlled studies to understand long-term results

Zhu, Y., Wang, Y., Jia, Y., Xu, J