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Shifts in microbial populations such as increased Prevotella and decreased Turicibacter along with disturbances in bile acid metabolism and lipid homeostasis, collectively highlight the gutliver axis as a central mechanism in cadmium-induced hepatotoxicity (4)
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Contribution of mitochondrial GSH transport to matrix GSH status and colonic epithelial cell apoptosis
These are made especially problematic due to the chemical solvents' ability to extract far greater amounts of the potentially toxic compounds than water
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