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In all the clinical studies, it demonstrated overall good tolerability, with no significant safety findings reported
The net effect: in serum or in cell culture media containing serum, free IGF-1 is rapidly bound and sequestered
ULK1 functions as a phosphatase in mitophagy by promoting FUNDC1 Ser17 phosphorylation to increase its LC3 binding ability, and upregulating ACT domain Ser108 phosphorylation in Parkin to activate it in an alternative pathway to PINK1 [173]
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