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Although rodent models provide important tools for AD research, it is difficult for these models to fully simulate the complex pathophysiological processes of human AD because of the significant differences in brain structure, metabolic characteristics, and immune responses between humans and experimental animals (Qian et al., 2024)
The result is improved wound stability, reduced scar formation, and stronger tissue remodeling, supporting its exploration in both trauma and cosmetic regenerative research
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In this context, Wan et al
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