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These discrepancies may be explained by the hypothesis that L-carnitine supplementation accompanied by high amounts of carbohydrate intake increases skeletal muscle OCTN2 expression through an insulin-mediated mechanism, thereby increasing skeletal muscle carnitine content [28, 29]
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We recommend the measurement of the Glx/NAA and GABA/Glx ratios, or other ways to normalize for neuronal loss, to both provide neuron specific measurements independent of volume changes as well as reduce methodological uncertainties
knowing TSH helps tailor the plan
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