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Given this unifying pathophysiology, GLP-1 RAs, with their powerful ability to act both centrally (controlling appetite and cravings) and peripherally (correcting systemic metabolic dysfunction, resolving hepatic steatosis and inflammation), to exert direct anti-carcinogenic effects, represent a truly multi-functional therapeutic tool
That is, EGT could couple GSH to reduce ferryl myoglobin, chelate transition metal iron to scavenge ROS/RNS and free radicals generated during IR, regulate proinflammatory cytokines like interleukin-1 (IL-1) and tumor necrosis factor- (TNF-), and ultimately prevent myocardial injury (Servillo et al., 2017a)
A-plaque associated (-AD1) and 3
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