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It is a synthetic 39-amino acid peptide that acts as an agonist at both the glucose-dependent insulinotropic polypeptide receptor (GIPR) and the glucagon-like peptide-1 receptor (GLP-1R), producing synergistic metabolic effects that exceed those of selective GLP-1 agonists in clinical trials
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On April 30, 2026, the FDA proposed to exclude semaglutide, tirzepatide, and liraglutide from the 503B bulks list entirely, citing no clinical need for large-scale compounding now that FDA-approved supply is stable
It is essential to document the temporal relationship between medication initiation and symptom onset, though causality should not be assumed without thorough investigation
As patients lose significant body weight on semaglutide, multiple cardiovascular parameters improve, including blood pressure, lipid profiles, and inflammatory markers
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