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The trans-3-hexenoyl group on the N-terminal tyrosine acts as a protective cap against dipeptidyl peptidase-IV (DPP-IV), the enzyme that cleaves native GHRH between residues 2 and 3 and generates the short plasma half-life that limits unmodified GHRH activity
On the other hand, TB-500 mediated cytoskeletal regulation can influence additional signaling networks because actin dynamics are closely linked to cell adhesion, migration signaling, and intracellular transport pathways [7]
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