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Blood 1998;92:1191-1198

Studies indicate the peptide may stabilize neuronal membrane function and protect against excitotoxicity associated with excessive glutamate signaling during brain injury

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To cope with this environmental pressure, it activates morphological switching, transitioning from the yeast to the hyphal form, thereby enhancing its survival and invasive capabilities

Once again, GHK-Cu appeared to mediate its effects through decreased TNF-alpha and IL-6 expression [11]