The dual effect is counterintuitive: Less acetaldehyde (liver-protective less toxic metabolite produced) Higher blood alcohol concentration (risky same drinks leave you more intoxicated) As principal investigator Wajahat Mehal, MD, professor of medicine at Yale, stated: "If these results are reproduced in humans, people using GLP-1 receptor agonists might be drinking an amount of alcohol that does not normally put them above the legal blood alcohol level, but because they are taking this drug, it does." Important caveat: This study was conducted in mice
Rather, it reveals baseline tendencies in appetite regulation and glucose metabolism
(5) In a particular study, researchers explored the theory that the peptide BPC-157 might potentially hasten wound healing compared to a control group
Inhibition of GLP-1R in the NTS (by Ex9) prevents Ex4 from executing its attenuation of alcohol-induced hyperlocomotion (Vallof et al., 2019b), which indicates that circulating GLP-1 may act via NTS to regulate alcohol responses
From there, youll get connected with a provider and learn more about your candidacy, including which GLP-1 is the best for you if approved