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Similarly, QUE enhanced the killing effect of DOX on P388 leukemia cells by inhibiting the expression of NF-B, p65, and Bcl-2 but by activating Bax and Caspase-3 (52)
Only 4.9% of the placebo group achieved this
METTL3 deposits m 6 A marks on CHAC1 transcripts, leading to their recognition by YTHDF2 and subsequent degradation
Preclinical studies have revealed complex neuroprotective mechanisms, including suppression of neuroinflammation, restoration of metabolic function, and activation of pro-survival pathways
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