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Abstract Background: The anti-inflammatory effect could be one of the mechanisms by which semaglutide reduces cardiovascular risk in patients with type 2 diabetes mellitus (T2DM) and/or obesity

Mechanistically, the NF-kB and cytokine effects should theoretically help, but lupus pathogenesis involves pathways (particularly type I interferon and complement activation) that may not be as responsive to GLP-1 signaling

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