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Other possible mechanisms to improve insulin sensitivity in NAFLD reported in the literature include the regulation of the acetyl-CoA/CoA ratio in mitochondria, modulation of the pyruvate dehydrogenase complex (PDHC) activity, altering the expression of glycolytic and gluconeogenic enzymes as well as the expression of genes associated with the insulin signaling cascade, and stimulation of the insulin-like growth factor-1 (IGF-1) axis and IGF-1 signaling cascade (55)

Additionally, in ME/CFS, the salivary glands undergo pathological changes, including mast cell accumulation and targeting by autoantibodies against muscarinic receptors [198]

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However, developers of GLP-2 therapies for chronic IBD will need to address prescriber concerns regarding long-term trophic effects and the necessity for mucosal dysplasia surveillance

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