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GHK-Cu stimulates synthesis of Type I collagen (structural support) and Type III collagen (tissue flexibility and repair)
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These high-order structures can compromise both the safety and efficacy of the final product
And GLP-1 can inhibite hepatocyte apoptosis which induced by NLRP3 through autophagy in mitochondria, weakening the intrahepatic inflammatory response, thereby inhibiting the transition from simple fatty liver to nonalcoholic steatohepatitis (NASH)
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