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In our study, treatment with AMPK inhibitor compound C rescued MG-induced cell death and apoptosis in RINm5F cells (Fig
A partir disso, foi possvel desenvolver medicamentos que mimetizam a ao ou inibiam a degradao desses "hormnios intestinais"
This could be due to pharmacokinetic changes, since drug distribution changes when body composition changes significantly
Given the expression of St14 (matriptase) in the intestinal crypt cells, we next determined the role of matriptase-PAR2 signaling for postprandial GIP regulation by using PAR2-K36E mutant mice
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