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CJC-1295 with DAC is engineered to last much longer (about a week) by binding to a blood protein
[1] Simultaneously, it suppresses glucagon secretion from pancreatic alpha cells, which helps prevent the liver from releasing excess glucose into the bloodstream
A study of 30 patients with type 2 diabetes treated for 8 weeks showed measurable reductions in CD62p (a marker of platelet activation) and PAC-1 (a marker of glycoprotein IIb/IIIa complex activation, the final common pathway of platelet aggregation)
Median [IQR] follow-up times in the two groups were of 642 [2231,414] days in the GLP-1 RA group and 529 [2071,131] days in the DPP-4i group
