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These are direct consequences of GLP-1 receptor activation in the gut

Extrinsic apoptotic pathways are activated by interactions between cell surface exposed death-inducing ligands such as FAS ligands (FASL) and tumor necrosis factor (TNF), and their cognate receptors Fas cell surface death receptor (FAS) and TNF receptor (TNFR) [110, 111]

Indeed, the process of adaptation to glutamine deprivation weakens the response to hypoxia, which normally strongly induces the expression of glycolytic enzymes 83

Neurogastroenterology and motility : the official journal of the European Gastrointestinal Motility Society

For standard review, the PDUFA date is set 1012 months after that acceptance