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Therefore, we concluded that BPC 157 affects on angiogenesis by activating the NO system through VEGF-A mediated AKT and p38/MAPK signaling pathways (Figure 5A and B)

The improvement in half-life can be attributed to two factors: Increase in molecular weight (approximately 60 kD), leading to an expansion in the fluid dynamic volume beyond the threshold of glomerular filtration in the kidneys