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Current research shows possible associations, not proof of cause and effect
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3b,c), the increase of [Ca 2+ ] i measured at the end-point was instead explained by TRPA1 channel activation in response to JWU-A021
These findings aligned with results from other analyses in our study, further confirming MKRN1 as a promising candidate therapeutic target for PPD and offers a foundational clue for future drug development efforts
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