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Altered prefrontal astrocytic GABA in postmortem brains of PTSD patients (postmortem human brain study) To investigate the molecular basis of elevated GABA levels observed in PTSD, we analyzed postmortem mPFC tissue from PTSD patients

Arg methylase inhibitors (AMIs), symmetric sulfonated urea, specifically inhibit PRMT activity and, in a rat model, cyclooxygenase-2 (COX-2) expression and suppress inflammation

Treatments of hypoxic cells with rapamycin lead to the degradation of HIF-1, which results in the increased inhibition of the expression of survivin and apoptosis in lung cancer cells

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