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This NAD + regulation is critical for clock-dependent acetylation, a modification that may influence the assembly and activity of CI 36
Niemann-pick C1-like 1 (NPC1L1) protein in intestinal and hepatic cholesterol transport
As a result, hepatocellular supplies of glutathione become exhausted and NAPQI is free to react with cellular membrane molecules, resulting in widespread hepatocyte damage and death, leading to acute hepatic necrosis
