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Nonerythropoietic erythropoietin-derived peptide suppresses adipogenesis, inflammation, obesity and insulin resistance
MCP-1 staining using immunohistochemistry and QuPath digital imaging showed that both male and female transgenic 5xFAD mice that received intranasal GHK-Cu had decreased staining intensity for MCP-1 in the frontal cortex and hippocampus (Figure 4) within tissues registering a positive stain for MCP-1
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Originally identified in human serum, GHK-Cu serves as a vital signal peptide that regulates extracellular matrix (ECM) remodeling, promotes fibroblast proliferation, and resets gene expression toward regenerative and antioxidant pathways
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