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However, 48 h post infection, HSV-1 positively regulated AMPK/Sirt1 axis showing an increase in Sirt1 activity and a reduction in the levels of acetylated p53 thus, promoting an anti-apoptotic state (Martin et al., 2014b)

3d and 3f, g), which might result in progressively greater loss of cell surface receptors over time, despite similar acute endocytosis rates

However, when the ROS level is high metabolic alterations of cellular constituents occurs related to oxidative damage to the cells [6, 10]