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These findings suggest that while FOXO4 is involved in the senescence pathway, it also plays a role in maintaining the viability of senescent Leydig cells by suppressing their apoptotic response, which could contribute to the accumulation of senescent cells [1]

Nevertheless, thymosin-4 has advanced into clinical studies for cardiac repair and corneal wound healing, demonstrating that non-classical signaling peptides can be developed therapeutically when endpoints are well defined

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